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Studies investigating muscle dysfunction in chronic fatigue syndrome have shown that patients exhibit an overreliance on this anaerobic metabolism even during low-level, everyday activities, leading to prolonged intramuscular acidosis and a slowed ability to clear protons from the tissue

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Immune cells like macrophages and neutrophils, while essential for pathogen clearance, can also contribute to lung injury when overactivated, highlighting the need for therapeutic strategies to modulate ferroptosis

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Interestingly, RipAYs enzymatic activity requires the activation by eukaryotic thioredoxins, which are highly abundant in plants during pathogen infection (Tada et al., 2008)