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Given the complex interrelationships among NAD + metabolism, mitochondrial function, and redox homeostasis, the uncontrolled supplementation of these elements may paradoxically lead to the exacerbation of oxidative stress or the impairment of cellular balance in individuals who are predisposed to such conditions

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Additionally, insulin resistance increases the number of neuritic plaques by affecting tau phosphorylation, widely considered a pivotal pathological hallmark in Alzheimers disease (Wegmann et al., 2021)