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This celebrated result was followed in 1983 by the work of Graeme Bell and colleagues at the Chiron Corporation (California), who cloned hamster and human cDNAs encoding preproglucagon [34, 35]

These disruptions trigger inflammation imbalance, immune suppression, and metabolic abnormalities, which may represent key potential mechanisms underlying various immune-related diseases and provide a theoretical basis for the association between environmental MPs exposure and skin immune system disorders