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In contrast, when placed under metabolic stress with increased demand on the -cell (induced by high fat feeding and a low dose of the -cell toxin, streptozotocin), glucose tolerance did become impaired, supporting the view that paracrine GLP-1 may be required to maintain glucose homeostasis during adaptation to metabolic stress (114)

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By simply telling the body to build more roads to the site of an injury, it allows for healing that is faster and stronger than nature usually allows

10.2147/copd.s226623 80 KulogluT.AydinS.ErenM

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