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However, the role of miR-335 in triggering lipid metabolism still remains unknown

In summary, TBI pathological effects on the BBB and subsequent excitotoxicity due to glutamate overload, lead to neuronal damage through oxidative stress

Additionally, liver cell injury and apoptosis can be promoted by lipid peroxidation, increased production of cytokines and ROS, as well as induction of Fas ligand, ultimately accelerating the transition to NASH

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Most patients (97%) were treated with one or more glucose lowering medications at baseline: metformin (76%), insulin (51%), SGLT-2 inhibitor (27%), sulfonylurea (29%), and DPP-4 inhibitor (23%)