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TB-500 is currently understood by FDA and WADA research as the N-terminally acetylated seven-residue thymosin beta-4 fragment Ac-Leu-Lys-Lys-Thr-Glu-Thr-Gln

The angiogenic action appears tied to T4s actin-binding motif: Philp et al., FASEB J 2003 demonstrated that this seven-residue site is essential for promoting endothelial migration and vessel formation

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